Damaged central nerves are thought to never regenerate. Our laboratory has reported a mechanism of how regeneration is inhibited. An extracellular sulfated glycan acts as a ligand for an enzyme-linked receptor expressed on the cell membrane of the axon terminal. This glycan regulates the autophagic flux into the cell, consequently regulating axonal elongation. The goal of my current research is to further elucidate the mechanisms underlying axonal elongation and the role of multiple types of sulfated glycans. I am investigating the effects of other intracellular signaling molecules whose activity is regulated by these enzyme-linked cell surface receptors on axonal elongation. Glycan codes are encoded in glycan structures, similar to how genetic codes are encoded in genetic material. However, it remains unknown whether living organisms have any innate system to intentionally generate these codes. In my future research, I want to seek answers to this question. I would like to master glycobiology and to contribute to society through the basic sciences.